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This open-access paper is pretty interesting with respect to mechanisms of action and how tapinarof differs from other stilbenes and AhR agonists: https://www.jidonline.org/article/S0022-202X(17)31543-9/full...

And, yeah, good point re coal tar. AhR was once thought to be a toxin or junk receptor that activated liver enzymes for clearance of environmental waste and other chemical byproducts. The constitutive androstane receptor (CAR) and pregnane X receptor (PXR) were, at one time, thought to be very similar. That might still be the case with respect to PXR and CAR, but I'm thinking that the way to bet is that there's more to them than was once thought...




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