> if you have a path in the upper bunk that gets broken you are screwed
Counter-factuals like this don't apply when talking about average probabilities. If you cross over, it's an identical graph with identical probabilities. idk, to me it seems really counter-intuitive that the opposite bunk's node would be easier to get to than the current bunk's node.
Suppose all the groceries raised the price of item X by exactly the same percent Y on the same day. At first glance this would look like perfect collusion in the data. But how do you know it wasn't just the supplier price going up by Y percent?
Suppose we took this line of thinking to its natural conclusion. If we wanted to be completely neutral, in theory we could have no male/female division at all, and just compete for the "best human" in each sport. But because of the vast biological differences between men and women, men would win every single time, hands down. This goes back to why do we have men and women divisions in the first place? Because we want a space for people without the vast advantages from male physiology to compete fairly with each other. Allowing for people who are technically "women" based on their reproductive anatomy, but have all the male physiological advantages otherwise, feels like it defeats the entire purpose from having separate women divisions.
What about a universal ranked system. For example, take tennis. There are already ranking systems so imagine a single one where all players regardless of gender/age/etc are all on a single list. Competitions will only allow you to play against someone within X rankings from you. So regardless of gender/age/etc, matches will always be reasonably fair and people move up and down based on what they win or lose.
So, now comes the part of whose the best DESC. Want to know the top female, just see the highest ranking one? Top person over 50? Under 20? Top person who isn't taking performance drugs? Top person who is? etc. You can get whatever top ranking you want. All matches are fair. Everyone can play.
This is actually the right answer though. Have one unrestricted category and then run the most popular retarded (I apologize, i can't find a better word, I have tried. It literally means poorer performance because of some specific identifiable physical trait in this case https://www.merriam-webster.com/dictionary/retard#h1) category next (which will often be women but could be men or something else in some instances) Relegate the rest to different events and promote/demote if popularity of some retardation (again, apologies) changes.
I think this is probably broadly true, but not always, maybe not even that commonly. Particularly in sports where raw power and endurance aren't the singular determining factor in the result. I'm optimistic that in some sports, if competition at a high-level started leaning into it, competitor performance from both would eventually converge in a way that makes the divide less clear. Some of those sports, by the nature of reduced exposure, just don't have remotely similar access or exposure among women, which is one of the factors that would change over time.
I'm thinking of climbing, skateboarding, where although speed, power, and endurance are factors, they're sort of relative factors compared to the person's bodyweight and how the routes/courses are set. Skateboard is much more divided for now than climbing as far as I can tell, but climbing is quite a lot closer, and the women's competition is generally more interesting to me to watch.
I feel like people aren't thinking enough about how much time each generation of new competitors has in terms of exposure to high-level athletes from the previous cohort, and how much of a compounding effect that can have. If you're clearly someone who'd be in the female category of sport, and you're divided into that category from the day you start, and the selection of people from the previous generation is 10% of the amount available among the other category, there's a very limited surface area for pushing harder.
In skateboarding, this pretty much meant anyone coming up 10 years ago had basically 2-5 notable figures who made anything of themselves skateboarding, and maybe half of them either stopped doing the sport or became reclusive. Ideally, you need to skate with the boys and compete with yourself and any other girl you can find. This period of time produced exactly what you'd expect; more women than in the previous generation got at least 50-75% better than the top competitors they would have looked up to. This compounds, and now those women are the inspiration for the current cohort, but they're only ever going to get as good or a bit better whichever rare individuals they have to compete with. Big fish, small pond.
In my opinion, we don't yet know how close those can get, and in some cases I'd like to see how that could change. In others, that possibility doesn't really come down to how driven or creative someone is, there's limited surface area for optimization and muscle growth, a clear dividing line where only mutant abnormalities would clear the gap at the highest level, since everyone on either side are already mutants.
Except sinusoids are special in that they are natural solutions to the Helmholtz wave equation. There's other problems too like square waves having infinite energy. This article might make sense to a mathematician or computer scientist but neglects the underlying physics of sound and waves.
Excellent point, lots and lots and lots and lots of physical objects are harmonic oscillators. That does have pretty fundamental grounding in physics.
I can think of lots of other places I'd use fourier analysis (at least qualitatively as with doing diffusion modeling in my head) but you're right that sinusoids are more physically "real" whereas being possible to represent in any basis set is more "valid" if that makes any sense.
Not quite sure what the right word is on this one, but I agree "real" kind of suggests real oscillators underlying the phenomena. Square waves are less physical because of discontinuities in both the signal and derivative; nature really doesn't care for discontinuities.
Frequency domain also makes the math really easy for linear, time-invariant operations, which (approximately) describe a lot of systems that exist in nature.
The Gibbs phenomenon, for example, falls out naturally from the IFT of a frequency response where all the frequencies above some cutoff are zero.
I'm curious how the square wave frequency domain would describe the Gibbs phenomenon -- I think you'd have harmonics of the fundamental square frequency showing up as if the system were nonlinear.
For anyone who's interested, there's a group of us dedicated to natural myopia reduction at reducedlens.org, which is a free and open-source fork of endmyopia. I've even started measuring my axial length to try and get better data on if this stuff actually works (only one measurement so far though, so nothing interesting yet).
The nature paper was pretty crazy. Basically because blue bends more than red (think of a prism), it also focuses a bit sooner. This phenomenon is known as longitudinal chromatic aberration, or LCA for short. This means if you're myopic, blue might be more blurry than red, and vice-versa if you're hyperopic. The researchers in the nature paper had participants watch a movie where they straight-up blurred the blue or red with software, in order to produce fake LCA signals. They found the participants axial lengths still shortened or lengthened anyway in response.
ReducedLens is absolute quackery! This is why I hate myopia discussions. Myopia is not reversible, if you think it is, you don't understand what axial elongation actually means.
Your eye's axial elongation is caused by the eye growing longer, which means *more cells in the eye.* Your eye grows in response to visual stimulus, without the brain's involvement. Your eye shape physically changes by *extra cell production* to cause axial elongation. This process is called emmetropization. It's extremely well understood. Peripheral defocus lenses and low dose atropine are proven to work in reducing speed of myopia progression because they reduce the stimulus causing the eye to elongate. Let me say that again: The way axial elongation happens is well understood, and we have known interventions to interfere with the growth stimulus. We know how to control myopia.
There is no known way to reverse this growth process. The only scientifically proven intervention for reducing myopia is surgery. There is no concrete evidence of for any other intervention, nor is there any other known mechanism for causing the eye to reshape itself to reduce axial elongation. For the eye to become truly less elongated, your body has to break down the extra cells in the eye and cause reshaping. An example of this is wound healing: your body breaks down the ECM with TMP activation. We know how the *opposite* works, the eye grows in response to overfocus to find focus. When too much is in focus in your peripheral vision (near work, holding things close to your face), the eye thinks it's over-focusing, and physically grows to find focus. Near work is the issue. Outdoor light probably isn't the issue, it's probably because when you're outside, you're not doing near work.
For those who do experience improvements, it's not from changes to the cellular structure of the eye. There are multiple temporary known mechanisms for your eye changing focus. One of them is the thickness of the choroid, a tissue layer in the back of the eye. Another is possibly muscular, your axial elongation is known to show variable diopters during the day (maybe +-0.25 but I don't know the actual variance). Studies do show fast-acting axial elongation reduction (minutes to hours) in response to defocus, but this is obviously not from the eye reshaping itself, so this is obviously not reversing myopia, this is just changing temporary variables in the eye. For the majority of people who report "reversing" myopia, it is usually a negligible change (2 diopters) which is easily explainable with these mechanisms, rather than the false belief it's actually changing the elongated eye shape. Ortho-K is another known temporary / superficial intervention.
The burden of proof of truly changing the axial elongation and fundamental structure of the eye cells is on you, and on the EndMyopia quacks. There are no studies that demonstrate significant diopter changes that would demonstrate the eye is reshaping itself to actually reverse myopia. You are welcome to find the studies on the Bates method and wearing undercorrected lenses yourself, they don't work and possibly make things worse.
Ok, here's proof of axial length changing following application of the method for years:
There was a member on the forums who was measuring his axial elongation while at the same time applying the reduced lens method. His result is shown in the following plot.[0] It is a significant improvement that can't be ignored, and can't be explained by day to day fluctuations or measurement error. So we know that at least some level of axial elongation can be reversed, and the idea is not complete quackery.
Also the reduced lens method has nothing to do with the Bates method, or undercorrection that leads to blur adaptation.
> "The only scientifically proven intervention for reducing myopia is surgery"
Which surgery reduces myopia? If you're thinking of LASIK then it doesn't change axial elongation.
Great. I would love to be proven wrong. My partial blindness from myopia related diseases will likely progress to full blindness as I age. I would love to be shown my worldview is wrong. For example, I didn't know anything about relative light color refractive differences until this HN post. I hope that science discovers a previously unknown mechanism or method for reducing axial elongation.
I also wish that eye doctors knew about the existing evidence. I wish that all opthamologists knew how emmetropization worked, I wish all lenses were peripheral defocused, and I wish more eye doctors prescribed low dose atropine to children, because the evidence is clear. And I sincerely hope that more eye doctors get sued for not using these tools in their practice. Ignoring science based evidence of myopia control in a field where you only need to know about 20 things is negligence.
Proving axial elongation is reversible is not done by a n=1 pet theory forum post measured in a home lab by someone who doesn't know what their choroid is. These forums are filled with people with mild myopia, not high myopes, who are "just starting my journey!" or "I had a small correction and I plateaued!" but are still zealously telling everyone else how to reduce their myopia. n=1 is fine for Reddit tier evidence, but without studies, it doesn't matter.
What is the source post of this? Is this chart really showing 0.1 millimeters of vitreous chamber depth change over 4 years? That almost sounds irrelevant, so maybe I'm missing something.
"Myopia is not reversible, if you think it is, you don't understand what axial elongation actually means."
There was a member on the forums who was measuring his axial elongation while at the same time applying the reduced lens method. His result is shown in the following plot.[0] It is a significant improvement that can't be ignored, and can't be explained by day to day fluctuations or measurement error. So we know that at least some level of axial elongation can be reversed, and the idea is not complete quackery.
Metals were very resource and labour intensive to make in those days, I don't think there was much making these for laughs and giggles. That also would would not explain why they are only found in the colder parts of the roman empire.
They’re actively developing to prevent this, though already it would require a compromised orb and orb operator, which would get blocked from the chain and all irises scanned since compromise removed from the chain. The bigger hurdle is the scanning of people who are dead or unconscious or animals trying to pass as human
Counter-factuals like this don't apply when talking about average probabilities. If you cross over, it's an identical graph with identical probabilities. idk, to me it seems really counter-intuitive that the opposite bunk's node would be easier to get to than the current bunk's node.